ocular hypertension

Cop-1-induced autoimmunization can protect glaucomatous retinas against ocular hypertension and prevent retinal ganglion cells from apoptosis.

结论Cop-1诱导的自体免疫反应可以对高眼压状态下的视网膜神经节细胞起到保护作用，减少视网膜神经节细胞的凋亡。

The mechanism of corticosteroid-induced ocular hypertension is increased aqueous outflow resistance.

类固醇诱导性高眼压的发生机制是房水流出阻力的增加.

Corticosteroid-induced ocular hypertension and glaucoma has been recognized for more than 50 years.

人们认识激素性高眼压和青光眼已经50多年了.

This study is designed to investigate the effects of transient ocular hypertension on retinal blood flow.

本研究主要观察瞬间性高眼压对视网膜血流量的影响.

Several variables have been identified as predisposing risk factors for steroid-induced ocular hypertension.

一些因素已经被确认为类固醇诱导性高眼压的主要危险因素.

Objective To investigate the efficacy of acute ocular hypertension of rabbits induced by cauterizing vortex veins.

目的探讨涡静脉烧灼法诱发兔急性高眼压的效果。

Protective effect of extract of crocus sativus on electroretinogram of rabbits with chronic ocular hypertension

藏红花提取液对慢性高眼压兔眼视网膜电图的保护作用

Objective To establish Cop-1-immunized models in rats with chronic ocular hypertension, to investigate the morphological changes that occur in all layers of the retina in glaucomatous rats, and to assess the role of autoimmunization induced by Cop-1 in protecting the glaucomatous rat's retina against ocular hypertension.

目的建立慢性高眼压大鼠Cop-1免疫模型，从形态学上观察Cop-1诱导的自体免疫对视网膜神经节细胞的影响。

Comparison of a non-preserved 0.1% T-Gel eye gel ( single dose unit) with a preserved 0.1% T-Gel eye gel ( multidose) in ocular hypertension and glaucomatous patients

无防腐剂0.1%T-GEL眼用凝胶（单剂）和含防腐剂0.1%T-GEL（多剂）对高眼压症和青光眼的疗效比较

Acute ocular hypertension of rabbits induced by cauterizing vortex veins

涡静脉烧灼诱发兔急性高眼压